You should predict infection by MRSA because the patient is colonized by MRSA. Most of beta lactamase antibiotics are ineffective in the treatment of MRSA.

It is a virulence factor, produced in 2 % strains of S. Aureus.

Suitable in sepsis, but only in combination with other antibiotics (bad penetration).

Fluconazole is more suitable than echinocandins, their MIC is rather high.

Cephalosporins and clindamycine are not effective against E. Faecalis. Sulbactam is useless. E. Faecalis isn’t a producer of beta lactamases. Inhibitor of beta-lactamases is unnecessary.

It is the most effective against Pseudomonas aeruginosa. Also, suitable for oral administration.

Fidaxomicin is suitable for the recurrence of colitis, it is the most effective antibiotic.

It is administered once daily.

It is supposed that infection can be caused by ESBL strain.

A slow infusion of vancomycin is a prevention of a red man syndrome, it is associated with a fast administration. Recommended time of the administration is 15 mg/ min.

There is an important horizontal gene transfer.

A Gram-positive rod living deep within skin follicles.

Therefore, it has a good penetration.

There is a high probability of efficiency against Pseudomonas aeruginosa

It is a new antimycotic agent with a wide spectrum of effectivity

This agent has frequent side effects.

Convulsions can be the adverse effect of imipenem.

Cephalosporins aren’t effective against anaerobic bacterias.

Getting rid of transient bacterias susceptible to alcohol, than getting rid of Cl. difficile by water and soap. Spores are resistant to alcohol.

Antibiotics listed below are destroyed by metalobetalactamase.

It is the first option antibiotic. Effective against MSSA.

Look at the Guidelines ESCMID and IDSA.

High recommendation, high-quality evidence - look at the Guidelines IDSA.

It is a natural resistance without an epidemiological impact.

Avibactam is an inhibitor of KPC. Beta lactamase antibiotics without avibactam, relebactam and some other ones are destroyed by KPC.

There is a risk of passing to the baby during childbirth. It can cause baby’s meningitis and sepsis.

An acute serious infection requiring rapid initiation of treatment.

Fasciitis, myositis, cellulitis.

Naturally resistant.

Therefore, it isnť suitable for septic patients.

Part of cell wall.

Could be a side effect, not a manifestation of an allergy.

According to international guidelines.

According to Stanford guidelines.

According to Stanford guidelines.

According to Stanford guidelines.

According to international guidelines.

According to Stanford guidelines.

According to international guidelines.

Tigecycline has a good penetration, it can be effective against producers of ESBL, therefore frequently used in hospitals.

According to international guidelines.

According to international guidelines.

Effective against staphylococcal infections.

Ceftaroline is suitable for MRSA, but its susceptibility must be tested in a microbiological lab

Strains of Enterococcus spp. are resistant to cephalosporins.

MRSA, Enterococcus faecium.

These are producers of AmpC beta-lactamases. Cephalosporins of the third generation aren’t suitable.

Penicillins and cephalosporins are destroyed by ESBL.

It is only used in the treatment of clostridial colitis.

It is a prodrug of mecillinam used for the infections of lower urinary tract.

It is neurotoxic and nefrotoxic.

It must have an effect in an intestine.

Cephalosporins of the third generation used in the treatment of gram-negative rods, suitable for the treatment of Pseudomonas aeruginosa infections.